prof. Birkmayer Laboratorien

Seznam publikací a studií

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MS Wordknihy-o-NADH-pro-laiky.doc (11 KB)MS Word
MS Wordpublikace-a-studie-o-NADH-I.doc (28 KB)MS Word
MS Wordpublikace-a-studie-o-NADH-II.doc (215 KB)MS Word
MS Wordpublikace-a-studie-o-NADH-III.doc (16 KB)MS Word
MS WordNADH-studie.doc (31 KB)MS Word
Adobe PDFkniha-NADH-the-energizing-coenzyme.pdf (133 KB)Adobe PDF

Knihy o NADH pro laiky

NADH The energizing Coenzyme. Prof.J.D.Birkmayer 1998

NADH Das Koenzyme für Gehirn. Prof.J.D.Birkmayer 1998

All about NADH. Prof J.D.Birkmayer 2000

Der Lebensnotwendige Zellwirkstoff NADH Koenzym 1. Prof. Hademan Bankfofer. 2008

NADH The biological Hydrogen. The Secret of Our Life Energy. Prof. J.D.Birkmayer 2009

Publikace a studie o NADH I

  1. Lehninger AL. “Vitamins and Coenzymes.“ Biochemistry, 2nd edition. The John Hopkins University School of Medicine, Worth Publishers, Inc. 1975; 337-342.
  2. Alberts B, Bray D, Lewis J, Raff H., Roberts K, Watson JD. “Energy Conversion: Mitochondria and Chloroplasts.“ Molecular Biology of the Cell, 3rd edition. Garland Publishing Inc. 1994; 653-720.
  3. Klingenberg M. “Pyridinnucleotide und biologische Oxydation,“ Zur Bedeutung der freien Nukleotide. II. Moosbacher Kolloquium, Springer Verlag, Heidelberg 1960; 82-114.
  4. Harden A, Young WJ. J. Physiol. 1905, 32.
  5. Garner RC, Farmer PB, Steel GT, Wright AS, eds. Human Carcinogen Exposure. Biomonitoring and Risk Assessment. Oxford: IRL Press, 1991.
  6. Grandjean P, ed. Ecogenetics, Genetic Predisposition to the Toxic Effects of Chemicals. London: Chapman and Hall, 1991: 3-18.
  7. Harns CC, Weston A, Willey JC, Trivers GE, Mann DL. “Biochemical and molecular epidemiology of human cancer: indicators of carcinogen exposure, DNA damage, and genetic predisposition.“ Environ. Health Perspect. 1987; 75: 109-119.
  8. Demopoulos HB et al. “The possible role of free radical reactions in carcinogenesis. “J. Environ. Path. Tox. 1980; 3: 273-303.
  9. Bankson DD, Kestin M, Rifai N. “Role of free radicals in cancer and atherosclerosis.“ Clin. Lab. Med. 1993; 13: 463-480.
  10. Halliwell B. “The role of oxygen radicals in human disease, with particular reference to the vascular System.“ Haemostasis 1993; 23(suppl): 118-126.
  11. Ueda K, Hayaishi 0. “ADP-Ribosylation.“ Ann. Rev. Biochem. 1985; 54: 73-100.
  12. Satoh MS, Poirier GG, Lindahl T. “NAD+-dependent repair of damaged DNA by human cell extracts.“ Biol. Chem. 1993, 268; 8: 5480- 5487.
  13. Badwey CW and Gerard RW. “Production of superoxide and hydrogen peroxide by an NADH oxidase in guinea pig polymorphonuclear leukocytes.“ J. Bio!. Chem. 1979; 254: 11530-11537.
  14. Grisham MB, Everse J. “The role of pyridine nucleotides in phagocytosis,“ in Everse: The Pyridine Nucleotide Coenzymes. Academic Press, Inc., New York, 149-278.
  15. Pryor WA. “Free radical reactions and their importance in biochemical systems.“ Fed. Proc. 1973; 32: 1862-1874.
  16. Tappel AL. “Lipid peroxidation damage to cell components.“ Fed. Proc. 1973; 32: 1870- 1874.
  17. Halliwell B, Gutteridge JMC. “Oxygen toxicity, oxygen radicals, transition metals and disease.“ Biochem. J. 1984; 219: 1-14. 21
  18. Cranton EM and Frackelton JR “Free radical pathology in age-associated diseases: Treatment with EDTA chelation, nutrition and antioxidants.“ J. Hol. Med. 1984; 6: 6-36.
  19. Halliwell B, Gutteridge JMC. “Role of free radials and catalytic metal ions in human disease: An overview.“ Methods Enzymol. 1990; 186: 1-85.
  20. Coon MJ. “Oxygen activation in the metabolism of lipids, drugs and carcinogens.“ Nuft Rev. 1978; 36: 319-328.
  21. Demopoulos HB, Pietronigro DD, Seligman ML. “The development of secondary pathology with free radical reactions as a threshold mechanism.“ J. Am. Coll. Tox. 1983; 2: 173-184.
  22. Halliwell B, Gutteridge JMC. Free Radicals in Biology and Medicine. Oxford: Ciarendon Press,1 985.
  23. Gutteridge JMC, Halliwell B. Antioxidants in Nutrition, Health and Disease. Oxford: Oxford University Press, 1994.
  24. Vrecko K, Birkmayer JGD, Krainz J. “Stimulation of dopamine biosynthesis in culture P12 pheochromocytoma cells by the coenzyme nicotinamide adenine dinucleotide (NADH).“ J. Neural. Transm. 1993; 5: 147-156.
  25. Gardier AM. Effects of Acute and Chronic NADH Administration on Peripheral and Central Norepinephrine and Dopamine Synthesis in the Rat. Birkmayer Institute for Parkinson Therapy, Intemal Lab Report No. 94070401.
  26. Kuhn W, Müller Th, Winkel R, Danielczik 5, Gerstner A, Häcker R, Mattem C, Przuntek H. “Parenteral application of NADH in Parkinson‘s disease: clinical improvement partially due to stimulation of endogenous levodopa biosynthesis.“ J. Neura!. Transm. 1996; 103: 1187-1193.
  27. Birkmayer W, Rieder P. Understanding the Neurotransmitters: Key to the Workings of the Brain. Springer Verlag: Wien-New York 1989.
  28. Koulousakis A, Nittner K. “Parkinson‘sche Erkrankung und Sexualfunktion.“ In: Fischer PA (ed.) Vegetativstörungen beim Parkinson-Syndrom. Roche, Basel 1984; 189-208.
  29. Ulm G, Suchy 1. “Drug treatment of Parkinson‘s disease with special reference to lisuride.“ In: Van Manesn J, Rinne UK (eds). Lisuride: A New Dopamine Agonist and Parkinson‘s Disease. Excerpta Medica Amsterdam, 1986: 55-63.
  30. Thorner MO, Vance ML. “Clinical aspects of dopamine in the regulation of human anterior pituitary function.“ In: Basic and Clinical Aspects of Neuroscience: The Role of Brain Dopamine. Springer Verlag 1989; 3: 19-29.
  31. Lieber CS. “Alcohol, liver, and nutrition.“ J. Am. Coll. Nutr. 1991, Dec 10(6): 602-632.
  32. Lieber CS. “Hepatic and metabolic effects of ethanol: pathogenesis and prevention.“ Annals of Medicrne 1994; 26(5): 325-
  33. Busheri N, Taylor J, Lieberman 5, Mirdamadi-Zonosi N, Birkmayer G, Preuss HG. “Oral NADH affects blood pressure, lipid peroxidation and lipid profile in spontanously hypertensive rats.“ J. Am. Coll. Nutr. 1997, in print.
  34. Baez S, Linderson Y, Segura-Aquilar J. “Superoxide dismutase and catalase enhance autoxidation during one-electron reduction of aminochrome by NADPH-cytochrome p-450 reductase.“ Biochemical and Molecular Medicine 1995: 54(1):12-18.
  35. Lamperth L, Dalakas MC, Dagani F, Anderson J, Ferrari R. “Abnormal skeletal and cardiac muscle mitochondria induced by zidovudine (AZT) in human muscle in vitro and in an animal model.“ Lab. Invest. 1991, Dec 65(6): 742-751.
  36. Bindoli A, Valente M, Cavallini L. “Prevention of lipid peroxidation by NADH(P)H in rat liver submitochondrial particles.“ Biochem. Int. 1987, Jul 15(1): 255-262. 22
  37. Pan SS, Akman SA, Forrest GL, Hipsher C, Johnson R. “The role of NAD(P)H: quinone oxidoreductase in mitomycin C- and porfiromycin-resistant HCT 116 human colon-cancer cells.“ Cancer Chemotherapy & Pharmacology 31(1): 23-31, 1992.
  38. Murray MF, Nghiem M, Srinivasan A. “HIV infection decreases intracellular nicotinamide adenine dinucleotide (NAD).“ Bioch. Bioph. Res. Comm. 1995; vol. 212, no. 1: 126-131.
  39. Klaidman LK, Mukherjee SK, Hutchin TF!, Adams JD. “Nicotinamide as a precursor for NAD+ prevents apoptosis in the mouse brain induced by tertiary-butylhydroperoxide.“ Neuroscience Letters 1996; 206: 5-8.
  40. Williams L. “Oxidative stress, age related neurodegeneration, and the potential for neurotrophic treatment.“ Cerebrovasc. Brain Metab. Rev. 1995; 7: 55-73. Chaykin 5. “Nicotinamide Coenzymes.“ Ann. Rev. Biochem 1962; 36: 149-170.
  41. Birkmayer JGD. “Stable, ingestable and absorbable NADH and NADPH therapeutic compositions,“ United States Patent No. 5.332.727. 1994.
  42. Birkmayer GJD, Birkmayer W. “Stimulation of the endogenous L-dopa biosynthesis - a new principle for the therapy of Parkinson‘s disease: the clinical effect of nicotinamide adenine dinucleotide (NADH) and nicotinamide adenine dinucleotidephosphate (NADPH).“ Acta Neurol. Scan. 1989; 126: 183-187.
  43. Birkmayer JGD, Vrecko C, Volc D, Birkmayer W. “Nicotinamide adenine dinucleotide (NADH)-a new therapeutic approach to Parkinson‘s disease, comparison of oral and parenteral application.“ Acta Neurol. Scand. 1993; 87 (Suppl 146): 32-35.
  44. Birkmayer CD, Birkmayer W. “The coenzyme nicotinamide adenine dinucleotide (NADH) as biological antidepressive agent experience with 205 patients.“ New Trends in Clinical Neuropharmacology 1991; 5: 75-86.
  45. Hazleton Europe Report No. 1174/1-1050: Final Report, “Birmadil (NADH): Single intravenous administration toxicity study in the rat,“ for Labor. Birkmayer & MEDINFO GesmbH, 1994.
  46. Hazleton Europe Report No. 1174/2-1050: Final Report, “Birmadil (NADH): Intravenous maximum tolerated dose (MTD) toxicity study followed by a 14 day fixed intravenous dose toxicity study in the beagle dog,“ for Labor. Birkmayer & MEDINFO GmbH, 1994.
  47. Birkmayer JGD. “The new therapeutic approach for improving dementia of the Alzheimer type.“ Ann. Clin. Lab. Sci. 1996; 26:1-9.
  48. Stocchi V, KoIb N, Cucchiarini L, Segni M, Magnani M, Fornaini G. “Adenine and pyridine nucleotides during rabbit reticulocyte matu ration and cell aging.“ Mechanisms of Ageing & Development 1987; 39: 29-44.
  49. Birkmayer W, Horsey Kiewic 0. “Der L-Dioxyphenylalanin (L-dopa) Effekt bei der Parkinson Akinese.“ Wien: Klm. Wochenschr. 1961; 73:787-788.
  50. Birkmayer W, Birkmayer JCD, Vrecko C, Paletta B, Reschenhofer E, Ott E. “Nicotinamide adenine dinucleotide (NADH) as edication for Parkinson‘s disease. Experience with 415 patients. New Trends in CIinical Neuropharmacology 4(1) 7-24, 1990.
  51. Fukuda K, Straus SE, Hickie 1 et al. “The chronic fatigue syndrome: a comprehensive approach to its definition and study.“ Internal Medicine 1994: 212: 953-959.

Publikace a studie o NADH II

  1. A close look at NAD biosynthesis. Mattevi, Andrea. 2006. 7, 2006, Nature Structural & Molecular Biology, Vol. 13, pp. 563-564. CoE1 allgemein.
  2. A conserved flavin-shielding residue regulates NO synthase electron transfer and nicotinamide coenzyme specificity. Adak, Subrata, et al. 2002. 2002, PNAS, Vol. 99, pp. 13516-1352.
  3. A pilot study of the effects of phytoestrogen supplementation on postmenopausal endometrium. Balk, Judith L., et al. 2002. 2002, J. Soc. Gynecol. Investig., Vol. 9, pp. 238-242.
  4. A Potential Role of the Curry Spice Curcumin in Alzheimer’s Disease. Ringman, John M., et al. 2005. 2005, Current Alzheimer Research, Vol. 2.
  5. A steady state analysis indicates that negative feedback regulation of PTP1B by Akt elicits bistability in insulin-stimulated GLUT4 translocation. Giri, Lopamudra, Mutalik, Vivek K and Venkatesh, KV. 2004. 2004, Theoretical Biology and Medical Modelling, Vol. 1.
  6. A very fast ion-pair reversed-phase HPLC method for the separation of the most significant nucleotides and their degradation products in human red blood cells. Stocchi, Vilberto, et al. 1987. 1987, Analytical Biochemistry, Vol. 167, pp. 181-190.
  7. Accelerated telomere shortening in response to life stress. Epel, Elissa S., et al. 2004. 2004, Proceedings of the National Academy of Sciences, Vol. 101, pp. 17312-17315.
  8. Adenosine Diphosphate Ribosylated Histones. Ord, Margery G. and Stocken, Lloyd A. 1977. 1977, Biochem. J., Vol. 161, pp. 583- 592.
  9. Age Related Changes in NAD+ Metabolism Oxidative Stress and Sirt1 Activity in Wistar Rats. Braidy, Nady, et al. 2011. 2011, PLOS ONE, Vol. 6. CoE1 allgemein.
  10. Aging, proteotoxicity, mitochondria, glycation, NAD+ and carnosine: possible inter-relationships and resolution of the oxygen paradox. Hipkiss, Alan R. 2010. 2010, Frontiers in Aging Neuroscience, Vol. 2, pp. 1-6.
  11. Ala-Rämi, Antti. 2003. Cellular energy state and calcium in myocardial substrate oxidation, ischemia and preconditioning. Academic Dissertation to be presented with the assent of the Faculty of Medicine, University of Oulu, for public discussion in the Auditorium L101 of the Department of Medical Biochemistry and Molecular Biology,. 2003. Dissertation.
  12. Albsmeier, Jennifer. 2004. Bedeutung endogener Inhibitoren der NO-Synthase für kardiovaskuläre Erkrankungen und ihre pharmakologische Therapie. Institut für Experimentelle und Klinische Pharmakologie, Arbeitsgruppe für Klinische Pharmakologie der Universität Hamburg. 2004. Dissertation.
  13. Althaus, Felix R., et al. 2005. Pharmakologie II. Pharmakologie II. 2005.
  14. An informatics search for the low-molecular weight chromium-binding peptide. Dinakarpandian, Deendayal, et al. 2004. 2004, BMC Chemical Biology, Vol. 4.
  15. Anderson, Bruce M. 1982. The Pyridine Nucleotide. 1982, Analogd of Pyridine Nucleotide Coenzymes, pp. 91-133.
  16. Angiotensin II Stimulates NADH and NADPH Oxidase Activity in Cultured Vascular Smooth Muscle Cells. Griendling, Kathy K., et al. 1994. 1994, Circ Res., Vol. 74, pp. 1141-1148.
  17. Anspach, Marie Christine. 2007. Überprüfung der Funktion der endothelialen NO-Synthase (ecNOS Typ III) bei Patienten mit leichtgradigem obstruktivem Schlafapnoe-Syndrom. Universitätsklinik der Ruhr-Universität Bochum. 2007. Dissertation.
  18. Anti-Aging-Medizin – Hoffnung oder Humbug? Kleine-Gunk, Bernd. 2007. 2007, Deutsches Ärzteblatt, Vol. 104, pp. 2054-2061.
  19. Anticancer Activities of Adenine Nucleotides in Tumor Bearing Hosts. Rapaport, Eliezer. 1993. 1993, Drug Development Research, Vol. 28, pp. 428-431.
  20. Antidepressant-like effect of nicotinamide adenine dinucleotide in the forced swim test in rats. Rex, Andre, Schickert, Ralph and Fink, Heidrun. 2004. 2004, Pharmacology, Biochemistry and Behaviour, Vol. 77, pp. 303-307.
  21. Applications of laser-induced fluorescence spectroscopy for the determination of NADH in experimental neuroscience. Rex, Andre and Fink, F. 2006. 2006, Laser Physics Letters.
  22. Arginine deficiency in preterm infants: biochemical mechanisms and nutritional implications. Wu, Guoyao, et al. 2004. 8, 2004, The Journal of Nutritional Biochemistry, Vol. 15, pp. 442-451.
  23. Asymmetric Dimethylarginine, an Endogenous Inhibitor of Nitric Oxide Synthase, Explains the “L-Arginine Paradox” and Acts as a Novel Cardiovascular Risk Factor. Böger, Rainer H. 2004. 2004, The Journal of Nutrition, pp. 2842-2847.
  24. Atherosklerotische Gefässerkrankungen und Stickstoffmonoxid (NO): Die wachsende Bedeutung von hoher Lebenserwartung und Übergewicht für die Klinik. Husmann, Marc, Keller, Mario and Barton, Matthias. 2007. 2007, Schweizerisches Medizinisches Forum, Vol. 7, pp. 1008-1011.
  25. Axon degeneration: Mechanisms and implications of a distinct program from cell death. Yan, Tingting, Feng, Yan and Zhai, Qiwei. 2010. 2010, Neurochemistry International, Vol. 56, pp. 529-534.
  26. Aydin, Jan. 2007. Skeletal Muscle Calcium Homeostasis during Fatigue - Modulation by Kinases and Mitochondria. Department of Physiology and Pharmacology; Karolinska Institutet, Stockholm, Sweden. 2007. Dissertation.
  27. Ayoub, Shereen. 2007. Modulation der neutralen Endopeptidase im humanen Zellkultursystem durch pflanzliche Drogenextrakte und Naturstoffe. Freie Universität Berlin. 2007. Dissertation.
  28. Bax/Bak-Dependent Release of DDP/TIMM8a Promotes Drp1-Mediated Mitochondrial Fission and Mitoptosis during Programmed Cell Death. Arnoult, Damien, et al. 2005. 2005, Current Biology, Vol. 15, pp. 2112-2118.
  29. Bechstest, Claudia. 2006. Nukleotide im Immunsystem – Untersuchung zur Bedeutung von ATP und NAD für die Lymphozytenfunktion anhand einer natürlich vorkommenden Mutation im murinen Purinorezeptor P2X7. Universitätsklinikum Hamburg-Eppendorf. 2006. Dissertation.
  30. Beneficial effects of antioxidants and L-arginine on oxidation-sensitive gene expression and endothelial NO synthase activity at sites of disturbed shear stress. Nigris, Filomena de, et al. 2003. 3, 2003, PNAS, Vol. 100, pp. 1420-1425.
  31. Benefits of Soy Isoflavone Therapeutic Regimen on Menopausal Symptoms. Han, Kyung K., et al. 2002. 2002, Obstet Gynecol, Vol. 99, pp. 389-394.
  32. Berger, Felicitas. 2003. NMN-Adenylyltransferase und NAD+-Kinase. Freie Universität Berlin. 2003. Dissertation.
  33. Bernitzki, Stefan. 2006. Referenzwerte gesunder Kinder und Jugendlicher für die Sauerstoffaufnahme in Ruhe, an der individuellen anaeroben Schwelle und bei Ausbelastung auf dem Laufbandergometer. Ruhr-Universität Bochum. 2006. Dissertation.
  34. Bioavailability of Reduced Nicotinamide adenine dinucleotide (NADH) in the Central Nervous System of the anaestetized Rat measured by Laser induced Fluorescence Spectroscopy. Rex, Andre, Hentschke, Mike-Percy and Fink, Heidrun. 2002. 2002, Pharmacology \& Toxycology, Vol. 90, pp. 220-225.
  35. Biochemical and Immunological Study on the Effects of Barley and its Components as Hypoglycemic Agents in Diabetic Rats. Yousef, Mokhtar I., et al. 2006. 2006, American Journal of Biochemistry and Biotechnology, Vol. 2, pp. 1-8.
  36. Bioenergetics and Death. Chernyak, B. V., et al. 2005. 2, 2005, Biochemistry, Vol. 70, pp. 294-301.
  37. Biosynthesis of retinoic acid by intestinal enzymes of the rat. Crain, F.D., Lotspeich, F.J. and Krause, R.F. 1967. 1967, Journal of Lipid Research, Vol. 8, pp. 249-254.
  38. Birkmayer, Georg D. 1997. NADH - The Energizing Coenzyme. 1997.
  39. Bound and unbound pyridine dinucleotides in normal and glucose-6-phosphate dehydrogenase-deficient erythrocytes. Canepa, Letizia, et al. 1991. 1991, Biochimica et Biophysica Acta, Vol. 1074, pp. 101-104.
  40. Brain NADH and jumping behavior in the rat. Shen, Zheng and Lin, Shu-Zhi. 1985. 1985, Life Sciences, Vol. 37, pp. 731-738.
  41. Braß, Anette. 2008. Bedeutung des P2X7 Purinorezeptors und von Ekto- ADP-Ribosyltransferasen (ARTs) für die Reifung und Funktion von antigenpräsentierenden Zellen der Maus (Mus musculus; Linnaeus, 1758). Universität Hamburg. 2008. Dissertation.
  42. Bugger, Heiko. 2006. Der Einfluß veränderter Arbeitslast auf die Regulation der respiratorischen Kapazität isolierter Mitochondrien. Albert-Ludwigs-Universität. 2006. Dissertation.
  43. Bünger, Frederic. 1999. Histomorphometrische Befunde am M. gluteus medius von Pferden ausgewählter deutscher Warmblutrassen -Selektionskriterium für die sportliche Veranlagung ? Veterinär-Anatomischen Institut der Veterinärmedizinischen Fakultät der Universität Leipzig. 1999. Dissertation.
  44. Bunse, Daniel. 2010. Referenzwerte gesunder Kinder und Jugendlicher für Laktat in Ruhe, an der individuellen ventilatorisch bestimmten Schwelle und bei Ausbelastung auf dem Laufbandergometer. Ruhr-Universität Bochum. 2010. Dissertation.
  45. Ca2+ controls slow NAD(P)H oscillations in glucose-stimulated mouse pancreatic islets. Luciani, Dan S., Misler, Stanley and Polonsky, Kenneth S. 2006. 2006, J. Physiol., Vol. 572.
  46. Ca2+, NAD(P)H and membrane potential changes in pancreatic ?-cells by methyl succinate: comparison with glucose. Heart, Emma, et al. 2007. 2007, Biochemical Journal, Vol. 403, pp. 197-20.
  47. Calorie restriction extends yeast life span by lowering the level of NADH. Lin, Su-Ju, et al. 2004. 2004, Genes \& Development, Vol. 18, pp. 12-16.
  48. Calorie Restriction Increases Muscle Mitochondrial Biogenesis in Healthy Humans. Civitarese, Anthony E., et al. 2007. 2007, PLOS Medicine, Vol. 4, pp. 485-494.
  49. Can the “brain-sparing effect” be detected in a small-animal model? Barbiro-Michaely, Efrat, et al. 2007. 2007, Medical Science Monitor, Vol. 13, pp. 211-219.
  50. Capsaicin Inhibits Preferentially the NADH Oxidase and Growth of Transformed Cells in culture. Morre, D. James, Chueh, Pin-Ju and Morre, Dorothy M. 1995. 1995, Proceedings of the National Academy of Sciences, Vol. 92, pp. 1831-1835.
  51. Characterization of Chromodulin by X-ray Absorption and Electron Paramagnetic Resonance Spectroscopies and Magnetic Susceptibility Measurements. Jacquamet, Lilian, et al. 2003. 2003, Journal of the Chemical Society, Vol. 125, pp. 774-780.
  52. Characterization of NAD Uptake in Mammalian Cells. Billington, Richard A., et al. 2008. 10, 2008, The Journal of Biological Chemistry, Vol. 283, pp. 6367-6374.
  53. Chromium in Health and Disease. Mahdi, Ghanim Salih. 2004. 2004, Kuwait Medical Journal, Vol. 36, pp. 38-41.
  54. Chromium Supplementation Does Not Improve Glucose Tolerance, Insulin Sensitivity, or Lipid Profile. Gunton, Jenny E., et al. 2005. 2005, Diabetes Care, Vol. 28, pp. 712-713.
  55. Chromium uptake by Saccharomyces cerevisiae and isolation of glucose tolerance factor from yeast biomass. Zetic, Vlatka Gulan, et al. 2001. 2, 2001, Journal of Bioscience, Vol. 26, pp. 217-223.
  56. Chromium, Glucose Intolerance and Diabetes. Anderson, Richard A. 1998. 6, 1998, Journal of the American College of Nutrition, Vol. 17, pp. 548-555.
  57. Chronic fatigue syndrome and mitochondrial dysfunction. Myhill, Sarah, Booth, Norman E. and McLaren-Howard, John. 2009. 2009, International Journal of Clinical and Experimental Medicine, Vol. 2, pp. 1-16.
  58. Chronic Fatigue Syndrome: A Review. Afari, Niloofar and Buchwald, Dedra. 2003. 2003, The American Journal of Psychiatry, Vol. 160, pp. 221-236.
  59. Chronic Mild Hypoxia Protects Heart-derived H9c2 Cells against Acute Hypoxia/Reoxygenation by Regulating Expression of the SUR2A Subunit of the ATP-sensitive K+ Channel. Crawford, Russell M., et al. 2003. 33, 2003, Journal of Biological Chemistry, Vol. 278, pp. 31444-31455.
  60. Chronische Erschöpfung und naturwissenschaftliche Erklärungsmodelle (Teil I). Höck, Anna Dorothea. 2009. 2009, CFS-aktuell.
  61. Circadian variation in urinary melatonin in clinically healthy women in Japan and the United States of America. Wetterberg, L., et al. 1979. 1979, Experientia, Vol. 35, pp. 416-419.
  62. Clinically Important Effects of Alcohol on Endocrine Function. Adler, Robert A. 1992. 1992, Journal of Clinical Endocrinology and Metabolism, Vol. 74, pp. 957-960.
  63. Coenzym-1 (NADH) - Eine wissenschaftlich bewiesene Anti-Aging-Substanz. Birkmayer, Jörg. 2006. 2006, Prevention and anti aging, Vol. 2, pp. 340-348.
  64. Coenzyme Nicotinamide Adenine Dinucleotide Hydrogen: Therapeutic Potential as a Cardioprotective Agent. Xu, Meng, et al. 2004. 2004, Journal of Tumor Marker Oncology, Vol. 19, pp. 241-250.
  65. Combinatory effects of phytoestrogens and 17ß-estradiol on proliferation and apoptosis in MCF-7 breast cancer cells. Schmidt, Simone, Michna, Horst and Diel, Patrick. 2005. 2005, Journal of Steroid Biochemistry \& Molecular Biology, Vol. 94, pp. 445- 449.
  66. Combined PEG liposomal doxorubicin and gemcitabine are active and have acceptable toxicity in patients with platinum- refractory and -resistant ovarian cancer after previous platinum-taxane therapy: a phase II Austrian AGO study. E., Petru, et al. 2006. 2006, Gynecologic Oncology, Vol. 102, pp. 226-229.
  67. Comparison of metabolic fates of nicotinamide, NAD+ and NADH administered orally and intraperitoneally; characterization of oral NADH. Kimura, Naoko, et al. 2006. 2006, Journal of Nutritional Science and Vitaminology, Vol. 52, pp. 142-148.
  68. Comparison of plasma and urinary levels of 2-hydroxyestrogen and 16?-hydroxyestrogen metabolites. Bradley, H. Leon, et al. 2006. 2006, Molecular Genetics and Metabolism, Vol. 87, pp. 135-146.
  69. Complementary and Alternative Medicine for Menopausal Symptoms: A Review of Randomized, Controlled Trials. Kronenberg, Fredi and Fugh-Berman, Adriane. 2002. 2002, Annals of internal medicine, Vol. 137, pp. 805-813.
  70. Consumption of nutritional supplements among adolescents: usage and perceived benefits. O’Dea, Jennifer A. 2003. 1, 2003, Health Education Research, Vol. 18, pp. 98-107.
  71. Contrasting effects of intracellular redox couples on the regulation of maxi-K channels in isolated myocytes from rabbit pulmonary artery. Thuringer, Dominique and Findlay, Ian. 1997. 1997, The Journal of Physiology, Vol. 500, pp. 583-592.
  72. Correlation of NADH and Ca2+ signals in mouse pancreatic acinar cells. Voronina, S., et al. 2002. 1, 2002, Journal of Physiology, Vol. 539, pp. 41-52.
  73. —. Voronina, S., et al. 2002. 2002, Journal of Physiology, Vol. 539, pp. 41-52.
  74. Coupling of histone deacetylation to NAD breakdown by the yeast silencing protein Sir2: Evidence for acetyl transfer from substrate to an NAD breakdown product. Tanny, Jason C. and Moazed, Danesh. 2001. 2001, PNAS, Vol. 98, pp. 415-420.
  75. Creatine kinase is physically associated with the cardiac ATP-sensitive k+ channel in vivo. Crawford, Russell M., et al. 2001. 2001, FASEB Journal.
  76. Cuntze, Cornelia. 2003. Untersuchungen zur Viskoelastizität von Tabletten mittels dynamisch-mechanischer Analyse. Mathematisch-Naturwissenschaftlichen Fakultät der Rheinischen Friedrich-Wilhelms-Universität Bonn. 2003. Dissertation.
  77. Curcuma, die Gelbwurz. Büechi, Samuel and Bolli, Richard. 2004. 4, 2004, Phytotherapie, pp. 26-31.
  78. Curcumin in Cell Death Processes: A Challenge for CAM of Age-Related Pathologies. Salvioli, S., et al. 2007. 2, 2007, Evidence- Based Complementary and Alternative Medicine, Vol. 4, pp. 181-190.
  79. Cytoplasmic Reduced Nicotinamide Adenine Dinucleotide Phospate-dependent and Microsomal Reduced Nicotinamide Adenine Dinucleotide Phospate-dependent Prostaglandin E 9-Ketoreductase Activities in Monkey and Pigeon Tissues. Lee, Sheng-Chung and Levine, Lawrence. 1974. 5, 1974, The Journal of Biological Chemistry, Vol. 249, pp. 1369-1375.
  80. D88A mutant of cytochrome P450nor provides kinetic evidence for direct complex formation with electron donor NADH. Umemura, Mariko, et al. 2004. 14, 2004, European Journal of Biochemistry, Vol. 271, pp. 2887-2894.
  81. Dayal, Eileen. 2005. Charakterisierung des Chemokinrezeptors CXCR3 in humanen proximalen Tubuluszellen der Niere. Albert- Ludwig-Universität Freiburg im Breisgau. 2005. Dissertation.
  82. Decline in skeletal muscle mitochondrial function with aging in humans. Short, Kevin R., et al. 2005. 2005, PNAS, Vol. 102, pp. 5618-5623.
  83. Decreased nocturnal plasma melatonin peak in patients with estrogen receptor positive breast cancer. Tamarkin, L., et al. 1982. 1982, SCIENCE, Vol. 216, pp. 1003-1005.
  84. Degradation Kinetics and Mechanism of RH1, a New Anti-Tumor Agent: A Technical Note. Jain, Neera, et al. 2007. 1, 2007, AAPS PharmSciTech, Vol. 8.
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  495. The Use of an Isoflavone Supplement to Relieve Hot Flushes. Jeri, Arturo R. 2002. 2002, The Female Patient, Vol. 27, pp. 35-37.
  496. Theoretical Study on the Antioxidant Activity of Curcumin. Sun, You-Min, et al. 2004. 2004, Chinese Journal of Chemistry, Vol. 22, pp. 827-830.
  497. Therapeutic effects of oral NADH on the symptoms of patients with chronic fatigue syndrome. Forsyth, Linda M, et al. 1999. 1999, Ann Allergy Asthma Immunol, Vol. 82, pp. 185-191.
  498. Therapeutic potential of NAD for neurological diseases. Ying, Weihai. 2007. 2007, Future Neurol., Vol. 2, pp. 129-132.
  499. Thomeczek, Helga Maria. 2001. Prophylaxe und Therapie des Jet-Lag Syndroms. Rheinisch-Westfälische Technische Hochschule Aachen. 2001. Dissertation.
  500. Topical application of NADH for the Treatment of rosacea and contact dermatits. Wozniacka, A., et al. 2003. 2003, Clinical and Experimental Dermatology, Vol. 28, pp. 61-63.
  501. Tostlebe, Mike. 2005. Disproportionalität der Aktivitäten der mitochondrialen Atmungskettenkomplexe im Myokard und in der Skelettmuskulatur im Alter. Martin-Luther-Universität Halle-Wittenberg. 2005. Dissertation.
  502. Transcriptional Control of Apolipoprotein A-I Gene Expression in Diabetes. Mooradian, Arshag D., Haas, Michael J. and Wong, Norman C.W. 2004. 2004, Diabetes, Vol. 53, pp. 513-520.
  503. Treatment of Alzheimer´s Disease with stabilized oral nicotinamide adenine dinucleotide: a randomized, double-blind study. Demarin, V., et al. 2004. 2004, Dru, Vol. 30, pp. 27-33.
  504. Treatment with reduced nicotinamide adenine dinucleotide (NADH) improves water maze performance in old Wistar rats. Rex, André, Spychalla, Markus and Fink, Heidrun. 2004. 2004, Behavioural Brain Research, Vol. 154, pp. 149-153.
  505. Trottmann, Matthias. 2004. Der Einfluss der Nikotinsäure auf die Expression von PPAR?, Scavenger Rezeptoren und ABCA-1 auf monozytoiden und hepatischen Zellen. Ludwig-Maximilians-Universität München. 2004. Dissertation.
  506. Two-photon autofluorescence dynamics imaging reveals sensitivity of intracellular NADH concentration and conformation to cell physiology at the single-cell level. Yu, Qianru and Heikal, Ahmed A. 2009. 2009, J Photochem Photobiol B., Vol. 95, pp. 46-57.
  507. Universality in intermediary metabolism. Smith, Eric and Morowitz, Harold J. 2004. 2004, PNAS, Vol. 101, pp. 13168-13173.
  508. Unlocking the Secrets of Longevity Genes. Sinclair, David A. and Guarente, Lenny. 2006. 2006, Scientific American, pp. 48-57.
  509. Uptake of nicotinamide adenine dinucleotide and excretion of its degradation pro-ducts by tissue culture cells. Seki, Shuji, et al. 1964. 1964, Acta Medica Okayama, Vol. 18.
  510. Use of Complementary and Alternative Medicine Among Persons With Diabetes Mellitus: Results of a National Survey. Yeh, Gloria Y., et al. 2002. 2002, American Journal of Public Health, Vol. 92, pp. 1648-1652.
  511. Vasomotor symptom relief by soy isoflavone extract tablets in postmenopausal women: a multicenter, double-blind, randomized, placebo-controlled study. Upmalis, David H., et al. 2000. 2000, Menopause, Vol. 7, pp. 236-242.
  512. Vehkavaara, Satu. 2001. Identification and Treatment of Endothelia Dysfunction and Cardiovascular Risk Markers in Disorders of Glucose Metabolism and in Postmenopausal Women. University of Helsinki. 2001. Dissertation.
  513. Venner, Monica. 2003. Akute interstitielle Pneumopathie beim Pferd - Experimentelle Induktion mit Perilla-Keton. Tierärztliche Hochschule Hannover. 2003. Dissertation.
  514. Verträglichkeit der konjugierten Östrogene und physiologische Form der HRT mit Schwangerschaftsöstrogenen. Huber, J. C. 2000. 2000, Journal für Menopause, Vol. 1, pp. 11-12.
  515. Vital imaging of H9c2 myoblasts exposed to tert-butylhydroperoxide – characterization of morphological features of cell death. Sardao, Vilma A., et al. 2007. 2007, BMC Cell Biology, Vol. 8.
  516. W.Addington, John. ENADA NADH: Trial Establishes Efficacy as Treatment for CFS. ENADA NADH: Trial Establishes Efficacy as Treatment for CFS.
  517. Water Maze Performance in Young Male Long-Evans Rats Is Inversely Affected by Dietary Intakes of Niacin and May Be Linked to Levels of the NAD1 Metabolite cADPR1. Young, Genevieve S., Jacobson, Elaine L. and Kirkland, James B. 2007. 2007, The Journal of Nutrition, Vol. 137, pp. 1050-10667.
  518. Weeß, H.-G., et al. 2001. Vigilanz, Einschlafneigung, Daueraufmerksamkeit, Müdigkeit, Schläfrigkeit...Die Messung müdigkeitsbezogener Prozesse bei Hypersomnien. Vigilanz, Einschlafneigung, Daueraufmerksamkeit, Müdigkeit, Schläfrigkeit...Die Messung müdigkeitsbezogener Prozesse bei Hypersomnien. 2001.
  519. Wehage, Edith Maria. 2003. Klonierung und funktionelle Charakterisierung des „long transient receptor channel 2“, LTRPC2, einem Mitglied der Familie kalziumpermeabler TRP-Kationenkanäle. Rheinisch-Westfälische Technische Hochschule Aachen. 2003. Dissertation.
  520. Weitere experimentelle Untersuchungen über den Catecholaminstoffwechsel bei extrapyramidalen Erkrankungen (Parkinson- und Chorea-Syndrom). Birkmayer, W. and Mentasti, M. 1967. 1967, Archiv für Psychiatrie und Zeitschrift f. d. ges. Neurologie, Vol. 210, pp. 29-35.
  522. Witt, Sören. 2004. Einfluss von Steigungstraining auf dem Laufband und unterschiedlichem Aufbautraining auf den Konditionserhalt bei Vielseitigkeitspferden. Institut für Tierzucht, Mariensee der Bundesforschungsanstalt für Landwirtschaft. 2004. Dissertation.
  523. X-ray induced L02 cells damage rescued by new anti-oxidant NADH. Liu, Fa-Quan and Zhang, Ji-Ren. 2003. 8, 2003, World Journal of Gastroenterology, Vol. 9, pp. 1781-1785.
  524. Zimmermann, Patricia Elaine. 2005. Proteomanalyse phänotypisch modulierter glatter Gefäßmuskelzellen. Technische Universität München. 2005. Dissertation.

Publikace a studie o NADH III

  1. Lehninger AL (1970) : Biochemistry, Worth Publisher Inc. 1970
  2. Klingenberg M. (1960) : Zur Bedeutung der freien Nucleotide. 11.Mobster Kolloquium Springer Verlag p82-114.
  3. Sauberlich HE (1987) : Nutritional Aspects of Pyridine Nucleotides, in Dolphin D, Poulson r, Avramovic O (editors) : Pyridine Nucleotide Coenzymes (Part B), John Wiley & Sons, Inc. 1987, pp. 599-626.
  4. Alberts B, et al (1983) : Molecular Biology of the Cell, Garland Publishing, 1983, pp. 491-495
  5. Vrecko K, Birkmayer JGD and Krainz J (1993) : Stimulation of dopamine biosynthesis in cultured PC12 phaeochromocytoma cells by the coenzyme nicotinamide adenine dinucleotide (NADH), J. Neur Trans. 5:147-156.
  6. Gardier M. (1994) Study Report to Labor Birkmayer, Department Pharmacology, University Paris Sud.
  7. Kaplan NO (1960) in Boyer PD, Lardy H and Myrbaeck K: The Enzymes Vol. 3, Ac Press New York p.156-172.
  8. Birkmayer JGD, Birkmayer W (1989): Stimulation of endogenous L-dopa biosynthesis- a new principle for the therapy of Parkinson’s disease. The clinical effect of nicotinamide adenine dinucleotide (NADH) and nicotinamid adenine dinucleotidephosphate (NADPH), Acta Neurol.Scand.126:183-187.
  9. Birkmayer W, Birkmayer JGD (1989): Nicotinamid-adenine-dinucleotide (NADH): The new approach in the therapy of Parkinson’s disease, Ann Clin Sci 19:38-43.
  10. Birkmayer W, Birkmayer JGD, Vrecko K and Paletta B (1990): The Clinical Benefit of NADH as Stimulator of Endogenous L-Dopa Biosynthesis in Parkinsonian Patients, Advances in Neurology Vol. 53: Parkinson’s Disease anatomy, Pathology and Therapy 545-549.
  11. Birkmayer W, Birkmayer JGD, Vrecko K, Paletta B, Reschenhofer E and Ott E (1990): Nicotinamide Adenine Dinucleotide (NADH) as medication for Parkinson’s disease: Experience with 415 patients, New Trends in Clin. Neuropharm. 4:7-24.
  12. Birkmayer W, Birkmayer JGD (1991): Nicotinamide Adenine Dinucleotide (NADH) as medication for depressed patients: Experience with 205 patients, New Trends in Clin. Neuropharm. 5:15-23.
  13. Birkmayer JGD, Vrecko K, Volc D and Birkmayer W. (1993) : Nicotinamide adenine dinucleotide (NADH) - a new therapeutic approach to Parkinson’s disease: Comparison of oral and parenteral application, Acta Neurol.Scand 87: Suppl.146:32-35.
  14. Birkmayer JGD (1995): Nicotinamide adenine dinucleotide (NADH) The new therapeutic approach for improving dementia of the Alzheimer type, submitted for publication.
  15. Passwater, Richard Ph.D. (1997) Paragraph 1 in the forward of the book called NADH The Energizing Coenzyme by Prof. Georg Birkmayer, M.D., Ph.D.
  16. Passwater, Richard Ph.D. (1997) Paragraph 2 in the forward of the book called NADH The Energizing Coenzyme by Prof. Georg Birkmayer, M.D., Ph.D.

NADH studie

Chronic Fatigue Syndrome (CFS)

  1. Forsyth LM, Preuss HG, MacDowell AL, Chiazze L Jr, Birkmayer GD, Bellanti JA. “Therapeutic effects of oral NADH on the symptoms of patients with chronic fatigue syndrome.” Ann Allergy Asthma Immunol. 1999 Feb;82(2):185-91.
  2. Santaella ML, Font I, Disdier OM. “Comparison of oral nicotinamide adenine dinucleotide (NADH) versus conventional therapy for chronic fatigue syndrome.” P R Health Sci J. 2004 Jun;23(2):89-93.

Parkinson’s Disease

  1. Birkmayer GJ, Birkmayer W. “Stimulation of endogenous L-dopa biosynthesis - a new principle for the therapy of Parkinson's disease. The clinical effect of nicotinamide adenine dinucleotide (NADH) and nicotinamide adenine dinucleotidephosphate (NADPH).” Acta Neurol Scand Suppl. 1989;126:183-7.
  2. Birkmayer JG, Vrecko C, Volc D, Birkmayer W. “Nicotinamide adenine dinucleotide (NADH) - a new therapeutic approach to Parkinson's disease. Comparison of oral and parenteral application.” Acta Neurol Scand Suppl. 1993;146:32-5.
  3. Birkmayer W, Birkmayer GJ, Vrecko K, Mlekusch W, Paletta B, Ott E. “The coenzyme nicotinamide adenine dinucleotide (NADH) improves the disability of parkinsonian patients.” J Neural Transm Park Dis Dement Sect. 1989;1(4):297-302.
  4. Birkmayer W, Birkmayer GJ. Nicotinamidadenindinucleotide (NADH): the new approach in the therapy of Parkinson's disease. Ann Clin Lab Sci. 1989 Jan-Feb;19(1):38-43.
  5. Birkmayer W, Birkmayer JG, Vrecko K, Paletta B. “The clinical benefit of NADH as stimulator of endogenous L-dopa biosynthesis in parkinsonian patients.” Adv Neurol. 1990;53:545-9. No abstract available.
  6. Dizdar N, Kagedal B, Lindvall B. “Treatment of Parkinson's disease with NADH.” Acta Neurol Scand. 1994 Nov;90(5):345-7.
  7. Kuhn W, Muller T, Winkel R, Danielczik S, Gerstner A, Hacker R, Mattern C, Przuntek H. “Parenteral application of NADH in Parkinson's disease: clinical improvement partially due to stimulation of endogenous levodopa biosynthesis.” J Neural Transm. 1996;103(10):1187-93.
  8. Swerdlow RH. “Is NADH effective in the treatment of Parkinson's disease?” Drugs Aging. 1998 Oct;13(4):263-8. Review.

Alzheimer’s Disease

  1. Birkmayer JG. “Coenzyme nicotinamide adenine dinucleotide: New therapeutic approach for improving dementia of the Alzheimer type.” Ann Clin Lab Sci. 1996 Jan-Feb;26(1):1-9.
  2. Demarin V, Podobnik SS, Storga-Tomic D, Kay G. “Treatment of Alzheimer's disease with stabilized oral nicotinamide adenine dinucleotide: A randomized, double-blind study.” Drugs Exp Clin Res. 2004;30(1):27-33.
  3. Rainer M, Kraxberger E, Haushofer M, Mucke HA, Jellinger KA. “No evidence for cognitive improvement from oral nicotinamide adenine dinucleotide (NADH) in dementia.” J Neural Transm. 2000;107(12):1475-81.


  1. Birkmayer JGD, Birkmayer W. “The coenzyme nicotinamide adenine dinucleotide (NADH) as biological antidepressive agent: Experience with 205 patients.” New Trends Clin Neuropharmacol. 1991;5:19–25.

Jet Lag

  1. Birkmayer GD, Kay GG, Vurre E. “Stabilized NADH improves jet lag-induced cognitive performance deficit.” Wien Med Wochenschr. 2002;152(17-18):450-4.

Interactions and Side Effects

  1. Birkmayer GD, Kay GG, Vurre E. [Stabilized NADH improves jet lag-induced cognitive performance deficit] Wien Med Wochenschr. 2002;152(17-18):450-4. German.
  2. Birkmayer JG, Nadlinger KF, Hallstrom S. “On the safety of reduced nicotinamide adenine dinucleotide (NADH).” J Environ Pathol Toxicol Oncol. 2004;23(3):179-94.
  3. Birkmayer JG, Nadlinger K. “Safety of stabilized, orally absorbable, reduced nicotinamide adenine dinucleotide (NADH): A 26-week oral tablet administration of NADH for chronic toxicity study in rats.” Drugs Exp Clin Res. 2002;28(5):185-92.

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